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Conclusions and Future Perspectives Influenza viruses systematically reprogram the host’s metabolic network to meet their needs for replication, survival, and propagation—a process that can be metaphorically termed “metabolic hostile takeover.” From enhancing glycolysis and suppressing oxidative phosphorylation to promoting anabolic pathways for lipids, amino acids, and nucleotides, influenza viruses skillfully manipulate the host cell’s resource supply system, effectively transforming it into a “factory serving viral replication.” This metabolic reprogramming not only provides the material foundation for viral replication but may also modulate host immune responses and cell fate, thereby further enhancing viral persistence and pathogenicity
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