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The following is a summary of documented tissue systems from peer-reviewed literature: This cross-tissue activity profile is directly attributable to the fact that its primary mechanisms — VEGFR2-driven angiogenesis and NO system modulation — are not tissue-specific

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Meanwhile, orthopedic surgeons were cataloguing a parallel revolution: synthetic peptides like BPC-157, originally derived from a protective protein in human gastric juice, were showing preclinical promise in accelerating tendon, ligament, and muscle repair—the very tissues endurance athletes stress on an almost daily basis