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3 Role of mitochondria in preventing/treatment of oxidative stress in PTSD Available data indicate that mitochondrial dysfunction, or suboptimal functioning, inherited or acquired, contribute to the manifestation of PTSD symptoms and can increase susceptibility to PTSD (for review, see, e.g., Preston et al., 2018
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Among lncRNAs, MALAT1, lincRNA-p21, TUG1, and H19 have been identified as key modulators: MALAT1 associates with UVB-induced senescence and photoaging in dermal fibroblasts and may have analogous roles in melanocytes, while lincRNA-p21 mediates p53-dependent apoptosis and cell cycle arrest to prevent malignant transformation 107
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