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This has been studied well in hepatocytes where chronic arsenic exposure induces hepatic DNA hypomethylation, which can potentially lead to aberrant gene expression and oncogenic growth in the liver, therefore suggesting a plausible mechanism of hepatocarcinogenesis (major cellular effects of arsenic are summarized in Figure 4) [18]
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[DOI] [PubMed] [Google Scholar] 236.Van Meter M, Mao Z, Gorbunova V, Seluanov A
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