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In this sense, some authors have described that mitochondrial dysfunction, increased oxidative stress, and chronic brain hypoperfusion contribute to the disruption of the BBB and cause damage to brain parenchymal cells [92,93]
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Nevertheless, even among the classical studies on trauma, three studies focused on mtDNA (66, 67, 170), although the fourth study found that serum IL-6 levels, inflammation and critical illness correlated with the levels of nDNA, not mtDNA (68)
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