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Our study found that chronic hypoxia promoted HIF-2α rather than HIF-1α entry into the nucleus, and nucleation of HIF-2α resulted in transcriptional expression of Acer2, a newly discovered downstream target of HIF that was reported in adipocytes [19, 24]

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This is also consistent with many previous works showing that interband transitions of CQWs are in-plane 5,6,45,46