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In the plasma, NAC undergoes redox exchange reactions with L-cystine, to generate plasma LC at a concentration capable of achieving the maximal rate of GSH synthesis in erythrocytes [69] and LC/NAC increase the rate of GSH biosynthesis in erythrocytes in vitro [70]

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In mice, a high-fat diet has also been shown to impair leptin signaling by attenuating STAT3 activation, thus contributing to leptin resistance [18].Leptin receptor activation engages intracellular proteins, such as protein tyrosine phosphatase1 B (PTP1B) and suppressor of cytokine signaling-3, that terminate receptor signaling

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